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Load management helps protect new tissue as it forms

Unlike direct GH administration, sustained GHS-R1a agonism preserves the pulsatile architecture of GH release a distinction of mechanistic importance in studies of GH secretory dynamics

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Research shows it enhances fibroblast migration to injury sites through the FAK-paxillin pathway, improves collagen fiber organization and cross-linking, promotes angiogenesis to improve nutrient delivery, and reduces inflammatory mediators while preserving beneficial healing signals